SARS

Mikovits, Judy A. @ https://noqreport.com/2021/05/30/dr-mercola-the-many-ways-in-which-covid-vaccines-may-harm-your-health/ - (edited) SARS-CoV-2 Spike Protein Engineered With HIV -
According to Mikovits, there’s evidence showing the SARS-CoV-2 spike protein was engineered by integrating HIV & XMRV proteins. XMRV stands for xenotropic murine leukemia virus-related virus, a human retrovirus that is very similar to endogenous retroviruses also found in other mammals.
XMRV has been linked to ME-CFS. HIV, which can cause AIDS, is another human retrovirus
(although as mentioned earlier, HIV does not appear to trigger AIDS all by itself. It needs a co-infection.)
'Our endogenous gamma-retrovirus is called human endogenous retrovirus-W (HERV-W). HERVW is all the way back in genesis in our original endogenous genome. It’s a gamma-retrovirus that expresses only the envelope, because in retroviruses, the envelope alone is enough to cause the disease.
That envelope protein is called syncytin.
They’re [now] calling it ‘spike protein’ just to throw us all off,' Mikovits says.
According to Mikovits, the SARS-CoV-2 virus was created by introducing a mutation into a molecular clone.
Vero E6 monkey tissues are known to be infected with SIV & other gamma-retroviruses.
The SARS-CoV-2 virus has markers suggesting it was grown in a Vero E6 cell line, she says.
'So syncytin is the gamma-retrovirus; it cross-reacts with the mouse & monkey gamma-retroviruses.
Monkeys, mice all have syncytin. Endogenous viruses express, especially during hormonal cycles.
When it’s expressed in the wrong place, like in the brain or the spinal cord, it’s long been associated with the inflammatory disease & destruction of the myelin sheet in multiple sclerosis (MS).
So, syncytin expressed it in the wrong place gives you the paralytics diseases.
We know Parkinson’s is associated with Type I interferon responses. We’re now starting to appreciate that there is low-level expression of our endogenous virome all the time & that in our innate immune response it’s trying to shape & educate our Type I interferon pathways …The final & biggest problem is these exosomes, because our body’s exosomes are like our cells’ response to express its regulatory RNAs, small inhibitory RNAs, long-chain non-coding RNA, which Ritchie Shoemaker has long associated with chronic Lyme & ME/CFS & the TGF-beta I pathway. TGF-beta I, that’s the master switch to turn on which Type I interferon, which [is needed for] myelopoiesis.
But these exosomes are packaging not only RNA that you’re making, but now you’ve dysregulated the methylation so you’ve woken up your endogenous virome & then syncytin is going to be expressed.'”